What role does LCT play in Blutzucker?

LCT encodes lactase-phlorizin hydrolase, the enzyme that breaks down lactose into glucose and galactose. This directly affects blood sugar: after dairy consumption, lactase-persistent individuals absorb glucose, raising postprandial blood glucose. In lactose-intolerant individuals (e.g., LCT-13910 C/T), undigested lactose causes bloating but no glucose spike. The role of LCT in fasting blood glucose is minor; its main impact is on post-meal glucose response. Some observational studies link lactase persistence to higher insulin secretion after milk intake, potentially influencing long-term diabetes risk, but evidence is moderate and confounded by diet. Consumer DNA tests often report LCT variants as part of nutrition panels, but they do not diagnose diabetes or replace glucose monitoring. For blood sugar management, overall diet, physical activity, and other genetic markers (e.g., TCF7L2) are more relevant. Evidence: human-moderate.