CRP (C-reactive protein) is a non‑specific inflammation marker that can rise due to acute infection, chronic inflammation, and psychological stress. The provided context does not include direct studies on CRP and stress. General evidence shows that stress activates the HPA axis and sympathetic nervous system, increasing pro‑inflammatory cytokines like IL‑6, which then stimulate hepatic CRP production. Genetic variants in the CRP gene have only small effects and are not clinically actionable. MyBody‑X DNA tests are educational, not diagnostic, for inflammation or stress. A more reliable approach is a fasting high‑sensitivity CRP (hs‑CRP) blood test, combined with stress reduction, sleep optimization, exercise, and an anti‑inflammatory diet (e.g., Mediterranean). Persistent hs‑CRP >2 mg/L warrants medical evaluation. Evidence for genetic CRP prediction is weak; focus on lifestyle and clinical measurement.
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